Hair loss affects millions of people, deeply undermining confidence and proving difficult to treat without identifying the underlying cause. When genetic predisposition, hormonal balance, nutrition and stress are evaluated together, an effective treatment plan can be created for the great majority.
The Hair Cycle and Mechanism of Loss
Each hair follicle cycles through growth (anagen), transition (catagen) and resting (telogen) phases. In a healthy scalp, about 85–90% of follicles are in anagen. When this ratio shifts — anagen shortens and telogen extends — daily shedding exceeds new growth and thinning begins.
Androgenetic alopecia (genetic loss) advances frontally-vertex in men and diffusely along the crown in women; the underlying mechanism is DHT (dihydrotestosterone) miniaturising hair follicles. Telogen effluvium presents as diffuse shedding after triggers — stress, fever, surgery, childbirth or nutritional deficiency. Both types differ in approach but both benefit from supportive clinical treatments.
Diagnosis: Understanding What Is Shedding
Effective treatment starts with the right diagnosis. Trichoscopy and follicular analysis image the hair cycle and follicle health. Laboratory testing investigates systemic causes: iron and ferritin (low ferritin is one of the most common drivers), B12 and folate, vitamin D, thyroid function and, where indicated, androgen profile. Addressing identified deficiencies in parallel with follicular support is essential.
Treatment Options
Hair mesotherapy: intradermal blends of vitamins, minerals, amino acids, coenzymes and growth factors feed the follicle, improve microcirculation and prolong anagen. A 4–8-session programme yields meaningful improvement.
PRP (Platelet-Rich Plasma): plasma drawn from the patient's blood and enriched in growth factors (PDGF, VEGF, EGF) is injected into the scalp; activates follicles and accelerates healing. Clinical evidence supports PRP in both androgenetic alopecia and telogen effluvium.
Exosome therapy: stem-cell-derived exosomes carry concentrated growth factors and microRNAs. Considered a more potent signalling load than PRP; useful in advanced cases or PRP non-responders.
Medical adjunct: approved drugs — minoxidil (topical/oral), finasteride or dutasteride — remain the foundation of treatment. Clinical procedures enhance their effect but do not replace them.
Course and Expectations
A typical course is monthly PRP or mesotherapy for 4–6 sessions, followed by maintenance every 2–3 months. Shedding typically slows over the first 2–3 sessions; new growth becomes visible by months 3–6. Realistic assessment requires at least a 6-month follow-up.
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